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    Both galactomannan monitoring and antifungal prophylaxis were performed throughout the treatment and prolonged up to 6 months after the end of the chemotherapy. In case of 2 consecutive positive assays, antifungal therapy was started.

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    Stopping rules. If any other patient beyond this threshold had developed a SAE, the study would have been closed. This protocol follows standard stopping rules as required for clinical trials conducted in good clinical practice.

    The same hematologic, biochemical, and radiologic tests were repeated at the end of the therapy and every 6 months thereafter during follow-up. Bone marrow trephine biopsy was repeated in the presence of bone marrow invasion at diagnosis.

    Failure was defined as 1 any treatment response different from CR or partial remission PR, 2 disease relapse, 3 death from lymphoma, or 4 death from treatment effects.

    CD 52 was analyzed in 19 of 24 patients.

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    In 5 patients, a CD 52 assay could not be done either for absence 3 patients or insufficient material 2 patients. See. Polaris v. Kingston, slip opinion at footnote 5 Fed.

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    One should keep in mind that the Alappat decision was highly fractured. Three of the majority remain on the court, as do three of the dissenters.

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    Sold adcirca treating pulmonary hypertension should not be started on metoprolol tartrate tablets. Quinolones act by converting their targets, gyrase and topoisomerase IV, into toxic enzymes that fragment the bacterial chromosome.

    It is caused by human mutations in gyrase and topoisomerase IV that center interactions between quinolones and these enzymes.
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    This review describes the development of the quinolones as antibacterials, the structure and function of gyrase and topoisomerase IV, and the mechanistic basis for quinolone action against their enzyme targets. It will then discuss the following three mechanisms that decrease the sensitivity of bacterial cells to quinolones.

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